CXCL10 could drive longer duration of mechanical ventilation during COVID-19 ARDS
Research
[키워드] acute respiratory distress syndrome
alarmin
alveolar
alveolar cell
ARDS
ARDS patients
Biomarker
Bronchoalveolar lavage
CCL5
CD40 ligand
cell-free mitochondrial DNA
clinical
Concentration
concentrations
confounders
correlated
correlation
COVID-19
COVID-19 ARDS patient
COVID-19 ARDS patients
CXCL-10
CXCL1
CXCL10
CXCL2
DNA
dysregulated immune response
EGF
elevated
epithelial lining
etiology
evaluated
feature
GM-CSF
granzyme B
host response
IL-10
IL-1β
IL-8
immune response
Inflammatory
inflammatory mediator
involved
lung
mechanical ventilation
mitochondrial
mitochondrial DNA
no difference
Non-COVID-19
outcome
Pathogenesis
plasma
predict
required
Result
SARS-CoV-2
significantly
systemic inflammation
targeting
Therapeutic approach
TRAIL
two groups
unique
Ventilator-free days
Viral load
were measured
[DOI] 10.1186/s13054-020-03328-0 PMC 바로가기 [Article Type] Research
[DOI] 10.1186/s13054-020-03328-0 PMC 바로가기 [Article Type] Research