S100A8 may govern hyper-inflammation in severe COVID-19
S100A8은 심각한 COVID-19에서 과염증을 지배할 수 있습니다.
Article
Published on
Journal: FASEB journal : official publication of the Federa [Category] SARS, 변종, 진단, 치료제,
Journal: FASEB journal : official publication of the Federa [Category] SARS, 변종, 진단, 치료제,
[키워드] accumulating
Activation
acute respiratory distress
acute respiratory distress syndrome
affected
ARDS
Biomarker
calcium
calcium-binding protein
cells
chronic myeloid leukemia
clinical information
Coagulation
coagulation cascade
coronavirus
coronavirus disease
Coronavirus disease 2019
COVID-19
COVID-19-associated ARDS
disease
disease severity
effective
Elimination
endotoxin
Evidence
extrinsic
form
herd immunity
homeostatic
hyper-inflammation
Hypothesis
Immunity
include
increased vascular permeability
Inflammation
Interaction
leukemia
Ligand
lung
molecular target
mortality rate
myeloid cells
myeloid-derived suppressor cells
overcome
pandemic
Pathogens
predict
reagent
receptor
recruitment
respiratory distress
Respiratory distress syndrome
S100A8
S100A9
severe COVID-19
severe forms of COVID-19
similarity
situation
syndrome
TLR4
to define
Toll-like receptor
Treatment
upregulated
upregulation
Vaccine
vascular permeability
[DOI] 10.1096/fj.202101013 PMC 바로가기 [Article Type] Article
[DOI] 10.1096/fj.202101013 PMC 바로가기 [Article Type] Article