IFN-γ protects from lethal IL-17 mediated viral encephalomyelitis independent of neutrophils
Research
[키워드] affecting
Autoimmune
blockade
CD4
CD4+ T cells
Central nervous system
clinical consequence
CNS
compared
complex
coronavirus
cytokine
Demyelination
detrimental
disease
downstream
effector molecule
Encephalomyelitis
expression
Factor
feedback loops
GM-CSF
IFN-γ
IFN-γ
IL-17
independent
Infected
inflammatory condition
inflammatory disease
Interaction
mechanism
memory
mice
morbidity
Mortality
murine
Neurotropic coronavirus
neutrophil
neutrophil infiltration
Neutrophils
not express
pathogenic
positive
PROTECT
provided
recipient
reduced
reducing
relative contribution
rescued
response
Result
survival
T cell
T cells
Th1
Th17
Th17 cell
tissue damage
Transcription
transcriptional regulation
transfer
upregulation
virus
was used
wild-type
[DOI] 10.1186/1742-2094-9-104 PMC 바로가기 [Article Type] Research
[DOI] 10.1186/1742-2094-9-104 PMC 바로가기 [Article Type] Research