SARS-CoV-2 deregulates the vascular and immune functions of brain pericytes via Spike protein
SARS-CoV-2는 스파이크 단백질을 통해 뇌 혈관주위세포의 면역 기능과 면역 기능을 완화합니다.
Article
[키워드] accompanied
accumulated
ACE2
ACE2 expression
acquisition
Actin
Activation
acute respiratory syndrome
acute respiratory syndrome coronavirus
acute respiratory syndrome coronavirus-2
angiotensin
Angiotensin-converting enzyme
angiotensin-converting enzyme 2
assays
binding
Brain
brain damage
calcium
cause
caused
cerebrovascular
cerebrovascular disorders
change
characterized
collagen
Comorbidity
contractile
coronavirus disease
coronavirus disease 19
Coronavirus-2
COVID-19
COVID-19 pathogenesis
Cytokines
deregulated
develop
disorder
enzyme
exacerbate
Express
expressing
expression
Fibronectin
fluctuations
function
functional
homolog
host cell
host cells
human ACE2
Human brain
hypoxia
immune
Immune cell
immune function
immune reaction
impair
infect
infection with SARS-CoV-2
Inflammation
inhibitory
intranasal
involved
lipid peroxidation
locus
Macrophage
mechanism
mechanisms
mice
MIF
Migration
morphology
Myofibrogenic transition
nasal mucosa
Neurological disorder
Neurological disorders
Neurovascular
Neurovascular interface
NF-κB
NOTCH3
nuclear
oxidative
pericyte
pericytes
phenotypic
pro-inflammatory cytokine
pro-inflammatory cytokines
promoted
Protein
Proteins
provide
reached
reactivity
receptor
recognize
Regulatory
Respiratory illness
S protein
SARS-CoV-2
SARS-CoV-2 S protein
SARS-CoV-2 S protein.
Severe acute respiratory syndrome
severe acute respiratory syndrome Coronavirus
shown
signaling pathway
spike
Stress
therapeutic interventions
transgenic mice
translated
transmembrane
triggered
Vascular
viral spike
was increased
[DOI] 10.1016/j.nbd.2021.105561 PMC 바로가기 [Article Type] Article
[DOI] 10.1016/j.nbd.2021.105561 PMC 바로가기 [Article Type] Article