oxidative damage

[키워드] ACHE activate Acute respiratory failure acute respiratory syndrome acute respiratory syndrome coronavirus acute respiratory syndrome coronavirus 2 adjuvant therapy age Anti-inflammatory anti-inflammatory effect antioxidant antitussive approved C-reactive protein caused clinical outcomes clinical trials Complication coronavirus coronavirus disease coronavirus disease-2019 correlated cough COVID-19 COVID-19 patient COVID-19 patients CRP cytokine Cytokine storm dependence disease dismutase drug Effect effective elevated ENhance enzyme Evidence FIBRIN glutathione Glutathione peroxidase headache help hypocoagulable Hypocoagulable effect IL-2 Immune-enhancing increase Increases interleukin lack Lactate lactate dehydrogenase LDH lead less malondialdehyde management mechanism mechanism of action medication moderate moderate to severe Mortality Multiple natural killer Necrosis old age oxidative damage Pain pathogen Pathogens patients patients with COVID-19 peroxidase Pneumonia polymerization PROTECT Pseudomonas aeruginosa psychiatric disorder Psychiatric disorders recent respiratory respiratory depression Respiratory failure SARS-CoV-2 SARS-COV-2 infection secretion severe COVID-19 patient severe COVID-19 patients severe pain severity Superoxide Superoxide Dismutase Support suppress T-cell T-cells therapeutic option therapy for COVID-19 thrombosis TNF-α tramadol Tramadol. Treatment treatment of COVID-19 patients tumor necrosis tumor necrosis factor tumor necrosis factor-alpha Venous Thromboembolism
[DOI] 10.1016/j.mehy.2020.110468 PMC 바로가기 [Article Type] Review