Influenza virus infection increases ACE2 expression and shedding in human small airway epithelial cells
Article
[키워드] Abstract
ACE2
ACE2 expression
activated
acute respiratory distress
acute respiratory syndrome
ADAM17
addition
Air-liquid interface
airway
airway epithelial cell
airways
amplify
Angiotensin-converting enzyme
baseline
caused
Cell
changes in
ciliated
cleavage
cleave
Co-infection
coronavirus
coronavirus disease
COVID-19
COVID-19 pandemic
distal
Donor
effort
epithelial cell
expressed
expression
goblet
IAV
increase
Infection
Influenza
influenza A
interactions
investigated
kDa
lung
Lung disease
lung homogenates
Lung injury
multiplicity of infection
Pathogenesis
pathogenic
predict
protease
protease TMPRSS2
proteolytic
receptor
required
respiratory viruses
Result
risk of COVID-19
SARS-CoV-2
SARS-COV-2 infection
severity
Single-cell RNA sequencing
Stat1
Support
syndrome
TACE
TMPRSS2
transcription factor
treatment of influenza
viral infections
virus infection
viruses
[DOI] 10.1183/13993003.03988-2020 PMC 바로가기 [Article Type] Article
[DOI] 10.1183/13993003.03988-2020 PMC 바로가기 [Article Type] Article