underlie
[용어속성] Action
COVID-19 Anosmia: High Prevalence, Plural Neuropathogenic Mechanisms, and Scarce Neurotropism of SARS-CoV-2?
COVID-19 ANOSMIA : SARS-COV-2의 높은 유병률, 복수의 신경 병원성 메커니즘 및 부족한 신경 프로즘?
Review
[키워드] absence
acute respiratory syndrome
acute respiratory syndrome coronavirus
analyzed
angiotensin
angiotensin-converting enzyme 2
Anosmia
apical surface
appear
causative pathogen
cause
Cell
cellular
Characteristics
convalescent
coronavirus
coronavirus disease
Coronavirus disease 2019
COVID-19
cytological
damages
examined
expression
in some
Infection
lack
mechanism
molecular
nasal
Nasal cavity
neurons
Neuropathology
ODs
olfactory
olfactory bulb
olfactory dysfunction
Olfactory epithelium
Pathogenesis
Perspective
Plural
Prevalence
priming
protease
receptor
respiratory
respiratory epithelium
respiratory virus
SARS-CoV-2
SARS-COV-2 infection
SARS-CoV-2 neurotropism
SARS-CoV-2 receptor
Serine
severe acute respiratory syndrome Coronavirus
severe acute respiratory syndrome coronavirus 2
Structure
susceptibility
target
transmembrane serine protease
transmembrane serine protease 2
underlie
Viral
viral infection
[DOI] 10.3390/v13112225 PMC 바로가기 [Article Type] Review
[DOI] 10.3390/v13112225 PMC 바로가기 [Article Type] Review
Mast cell activation symptoms are prevalent in Long-COVID
비만세포 활성화 증상은 Long-COVID에서 만연
Article
[키워드] Activation
age
Analysis
assessment
caused
cell activation
Control
COVID-19
COVID-19 symptoms
effective therapy
Facebook
fatigue
females
group
hyper-inflammation
Increased
life
long-COVID
mast cell
mast cell activation.
Mast cells
mechanism
pathophysiology
Patient
prevalent
Quality of life
recruited
reported
SARS-COV-2 infection
severity
subject
Support
Symptom
Symptoms
syndrome
Treatment
underlie
[DOI] 10.1016/j.ijid.2021.09.043 PMC 바로가기 [Article Type] Article
[DOI] 10.1016/j.ijid.2021.09.043 PMC 바로가기 [Article Type] Article
Differential mortality of infectious disease in Italian polities: COVID-19, past plague epidemics, and currently endemic respiratory disease
Research article
[키워드] analyses
Analysis
Bubonic plague
Coronavirus disease 2019
COVID-19
COVID-19 mortality
COVID-19 pandemic
criterion variable
Curve
disease
Endemic
Endemic disease
Epidemic
epidemic disease
Epidemics
Evidence
Factors
Foreign
GDP
greater
impacted
include
indicated
Infectious disease
Italian
Italian population dynamics
Italian regions
Italy
morbidity and mortality
Mortality
mortality rate
outbreak
outcome
pathogen
persist
plague
predictors
Prevalence
provide
Respiratory disease
South
Spread
statistical
statistical parameter
tested
underlie
[DOI] 10.1016/j.meegid.2021.105081 [Article Type] Research article
[DOI] 10.1016/j.meegid.2021.105081 [Article Type] Research article
Direct SARS-CoV-2 infection of the human inner ear may underlie COVID-19-associated audiovestibular dysfunction
Article
[키워드] ACE2
angiotensin-converting enzyme 2
caused
Cell
cell types
cellular
cofactor
confocal microscopy
Coronavirus disease 2019
COVID-19
COVID-19 patient
demonstrated
Direct
disease
Dizziness
dysfunction
Express
expression
hearing loss
in vitro
in vitro model
infect
Infection
infection rate
Infectious disease
molecular
molecular mechanism
novel coronavirus SARS-CoV-2
Organoid
organoids
pandemic
patients with COVID-19
pluripotent stem cells
precursor
problem
protease
receptor
required
Result
SARS-CoV-2
SARS-CoV-2 entry
SARS-CoV-2 entry factors
SARS-COV-2 infection
SARS-CoV-2 virus
Schwann cell
serine 2
stem cells
susceptible
Symptom
Symptoms
target
these symptoms
tissue
tissues
TMPRSS2
transmembrane
two-dimensional
underlie
vascular disease
Virology
virus entry
with COVID-19
[DOI] 10.1038/s43856-021-00044-w PMC 바로가기 [Article Type] Article
[DOI] 10.1038/s43856-021-00044-w PMC 바로가기 [Article Type] Article
Clinical practices underlie COVID-19 patient respiratory microbiome composition and its interactions with the host
임상 관행은 COVID-19 환자의 호흡기 미생물군유전체 구성 및 숙주와의 상호작용의 기초가 됩니다
Article
[키워드] antibiotic
association
Bacteria
changes in
clinical
clinical microbiology
Community
condition
confounder
confounders
contribute
contributing to
Control
COVID-19
COVID-19 patient
COVID-19 patients
Disease progression
driving
Evidence
exacerbated
explain
Host
ICU
identify
immune cells
immune response
immune responses
Infection
inflammatory markers
inflammatory phenotype
intensive care
intensive care unit
Interaction
Lower respiratory tract
Lungs
mechanical ventilation
Microbiome
microbiome composition
microbiota
oxygen
oxygen support
pathology
physical
physically
proinflammatory
reduced
Research
respiratory
respiratory tract
SARS-CoV-2 viral load
Sequencing
severe COVID-19 patients
severe disease
Single-cell transcriptomics
Treatment
underlie
understanding
upper respiratory tract
Variation
Viral
Viral load
with COVID-19
[DOI] 10.1038/s41467-021-26500-8 PMC 바로가기 [Article Type] Article
[DOI] 10.1038/s41467-021-26500-8 PMC 바로가기 [Article Type] Article
Structural and Functional Analysis of Female Sex Hormones against SARS-CoV-2 Cell Entry
Article
[키워드] ACE2
ACE2 receptor
alveolar
cells
COVID-19
disrupt
ENhance
Estrogen
estrogenes
Evidence
female
females
glycosylation
hormone
human ACE2
human ACE2 receptor
Interaction
male
mechanism
molecular basis
Protein
reduced
SARS-CoV-2
Severe infection
sex hormone
Sex hormones
Spike protein
susceptible
the SARS-CoV-2
the SARS-CoV-2 virus
therapy
underlie
variety
[DOI] 10.3390/ijms222111508 PMC 바로가기 [Article Type] Article
[DOI] 10.3390/ijms222111508 PMC 바로가기 [Article Type] Article
X-Linked TLR7 Deficiency Underlies Critical COVID-19 Pneumonia in a Male Patient with Ataxia-Telangiectasia
X-연관 TLR7 결핍은 운동실조-모세혈관확장증 남성 환자의 심각한 COVID-19 폐렴의 원인이 됩니다
Case Reports
[키워드] antibody
Antibody deficiency
Asymptomatic
Ataxia
ataxia-telangiectasia
ATM
autoantibodies
autoantibody
candidate gene
clinical manifestations
conditions
coronavirus disease
Coronavirus disease 2019
COVID-19
COVID-19 immune response
COVID-19 pneumonia
Critical
critical COVID-19
deficiencies
deficiency
Deleterious
Diagnosis
disease
evaluated
exhibit
Exome
genetic cause
genetic defect
genetic defects
high risk
homozygous
identify
IFNs
IgM
immune response
Immunity
Impact
inborn errors of immunity
individual
interferons
investigated
life-threatening
mechanism
Mild symptom
mild symptoms
Mutation
neurological signs
objective
Patient
patients evaluated
Pneumonia
Primary Immunodeficiency
radiosensitivity
Result
SARS-COV-2 infection
Sequencing
serum levels
specific antibodies
specific antibody
supplementary material
syndrome
telangiectasia
the patient
TLR7
TLR7 gene
Treatment
Type I IFN
type I interferon
type I interferons
underlie
understanding
unique
variant
virus
was performed
were measured
[DOI] 10.1007/s10875-021-01151-y PMC 바로가기 [Article Type] Case Reports
[DOI] 10.1007/s10875-021-01151-y PMC 바로가기 [Article Type] Case Reports
Allotypic variation in antigen processing controls antigenic peptide generation from SARS-CoV-2 S1 spike glycoprotein
Research Article
[키워드] acute respiratory syndrome
Allotype
allotypes
Analysis
analyzed
antigen processing
antigenic peptide
antigenic peptides
approach
breadth
Cell
contribute
Control
coronavirus
coronavirus disease
COVID-19, coronavirus disease 19
Critical
cytotoxic
determinant
Endoplasmic reticulum
enzyme
enzyme mechanism
Epitopes
ER, endoplasmic reticulum
ERAP1
ERAP1, endoplasmic reticulum aminopeptidase 1
genetic variability
glycoprotein
hallmark
heterogeneity
histocompatibility complex
HLA
HLA, human leukocyte antigen
immune response
immune system gene
Immunity
individuals
Infection
Ligand
mechanism
MHC
MHC, major histocompatibility complex
Pathogens
PCA, principal component analysis
peptide
peptides
Population
precursor
produced
producing
Proteolytic processing
proteomic
proteomics
PSM, peptide-spectrum match
reaction
regulate
response
SARS-CoV-2 S1 spike
SARS-CoV-2, Severe acute respiratory syndrome coronavirus 2
sequence
severity
significant difference
SNPs
spike glycoprotein
suggested
T-lymphocyte
trimming
underlie
vaccination
Variability
Variation
while
[DOI] 10.1016/j.jbc.2021.101329 PMC 바로가기 [Article Type] Research Article
[DOI] 10.1016/j.jbc.2021.101329 PMC 바로가기 [Article Type] Research Article
Functional Effects of Cardiomyocyte Injury in COVID-19
COVID-19에서 심근세포 손상의 기능적 효과
Cellular Response to Infection
[키워드] absence
acute respiratory syndrome
acute respiratory syndrome coronavirus
acute respiratory syndrome coronavirus 2
Affect
Analysis
Blood
cardiac disease
Cardiomyocytes
Cell
clinical
Clinical data
Clinical finding
Clinical findings
Comorbidities
Comorbidity
Computational biology
contractile
contractile apparatus
coronavirus
COVID-19
COVID-19 patient
COVID-19 patients
Culture
decrease
decreased expression
develop
disease
disorganization
dysfunction
echocardiography
Effect
elevated
Elevation
expression
Extracellular
healthy
heart
heart disease
heart tissue
hearts
hospitalized patient
hospitalized patients
identify
IL-1β
IL-10
IL-1β
IL-6
increase
infect
Infection
Injury
interleukin
Interleukins
Laboratory
machine learning
mechanism
Mount Sinai
Mount Sinai Health System
mRNA
multinucleated cells
multiple organs
NL63
observation
Patient
physiological
physiological change
physiological effect
physiological effects
pluripotent stem cell
Protein
reduced
respiratory
resulting
SARS-CoV-2
SARS-COV-2 infection
severe acute respiratory syndrome Coronavirus
subject
subset
suppresse
System
Treatment
Trigger
triggers
troponin
troponin I
underlie
ventricular
ventricular ejection
Viral
viral infection
virus
virus infection
[DOI] 10.1128/JVI.01063-21 PMC 바로가기 [Article Type] Cellular Response to Infection
[DOI] 10.1128/JVI.01063-21 PMC 바로가기 [Article Type] Cellular Response to Infection