wound healing

[키워드] ACE2 Activation acute respiratory syndrome acute respiratory syndrome coronavirus acute respiratory syndrome coronavirus 2 age age and sex Antiviral antiviral response antiviral responses CD8A cells chemokine chemokines chemokines CXCL9 clinical manifestation clinical manifestations contribute coronavirus Course Culture cultures CXCL9 CXCR3 Cytotoxic T cell Cytotoxic T cells days post infection deficiencies deficiency demonstrated dependent on disease severity elapsed elevated examined expression Factor females Gene Expression Genes genomic granzyme B HAE Host host response host responses Host transcriptional response human airway epithelial humoral immune responses IFIT1 immune responses in vivo increase in individual induce Infection Inflammation inhibitor inhibitors interferon interferon signaling interferon-stimulated gene interferon-stimulated genes male mechanism nasopharyngeal natural killer negative controls neutrophil NF-κB nuclear OAS1 Patient PCR-confirmed populations profile Protein Proteins receptor reduced reduction in relative replicated respiratory reveal RNA sequencing SARS-CoV-2 SARS-COV-2 infection severe acute respiratory syndrome Coronavirus Sex shotgun shown Signaling specimen Swab T cells T helper Th1 transcript Transcription transcripts up-regulation Viral Viral load wound healing
[DOI] 10.1371/journal.pbio.3000849 PMC 바로가기 [Article Type] Research Article