Abstract
The spectrum of pulmonary parenchymal and vascular pathologies related to the COVID-19 have emerged. There is evidence of a specific susceptibility related to thrombotic microangiopathy in situ and a complex immune-inflammatory cascade, especially in the pulmonary vascular bed. The potential to lead to transient or self-correcting sequelae of pulmonary vascular injury will only become apparent with longer-term follow-up. In this review, we aimed to present the findings in a group of patients with severe pneumonia due to covid-19 complicated by acute pe documented by chest angiography, who during a follow-up of more than 3 months with oral anticoagulant met clinical, hemodynamic, and imaging criteria of chronic thromboembolic pulmonary hypertension. We present a brief review of the epidemiology, pathophysiology, clinical findings, comorbidities, treatment, and imaging findings of chronic thromboembolic pulmonary hypertension as a sequel of severe post-covid-19 pneumonia; and compared and discussed these findings with similar reports from the medical literature.
【초록키워드】 COVID-19, Treatment, pathology, Epidemiology, susceptibility, Comorbidities, Clinical findings, pathophysiology, Chest, Patient, Follow-up, severe pneumonia, hemodynamic, Anticoagulant, pulmonary hypertension, Evidence, criteria, Vascular injury, thromboembolic, Vascular, complex, thrombotic, cascade, parenchymal, 【제목키워드】 review, pulmonary, chronic, Sequel,