Highlights • Neurological symptoms are dissociated from pulmonary involvement in COVID-19. • Despite disease heterogeneity, COVID-19 patients shared a single altered CSF proteomic profile. • Deregulated CSF proteomic pathways related to the innate immune system and hemostasis. • Mild and severe COVID-19 patients with neurological symptoms display systemic and central nervous system hyper-inflammation. • Disease severity is associated with increases in CSF IL6. • Neuroimaging abnormalities are connected to elevated CSF IL6 and TNF α levels.
All Keywords
【저자키워드】 COVID-19, Inflammation, coronavirus, Neuroinflammation, Neuro-infectious diseases,
【저자키워드】 COVID-19, Inflammation, coronavirus, Neuroinflammation, Neuro-infectious diseases,