Abstract Recent evidence is focusing on the presence of a hypercoagulable state with development of both venous and arterial thromboembolic complications in patients infected with SARS-CoV-2. The ongoing activation of coagulation related to the severity of the illness is further characterized by thrombotic microangiopathy and endotheliitis. These microangiopathic changes cannot be classified as classical disseminated intravascular coagulation (DIC). In this short review we describe the interaction between coagulation and inflammation with focus on the possible mechanisms that might be involved in SARS-CoV-2 infection associated coagulopathy in the critically ill.
All Keywords
【초록키워드】 Inflammation, SARS-COV-2 infection, severity, Endotheliitis, Coagulation, Coagulopathy, Critically ill, Patient, change, mechanism, Disseminated intravascular coagulation, Evidence, Interaction, hypercoagulable, Activation, thrombotic, recent, venous, classical, involved, characterized, infected with SARS-CoV-2, thromboembolic complication, 【제목키워드】 the SARS-CoV-2,
【초록키워드】 Inflammation, SARS-COV-2 infection, severity, Endotheliitis, Coagulation, Coagulopathy, Critically ill, Patient, change, mechanism, Disseminated intravascular coagulation, Evidence, Interaction, hypercoagulable, Activation, thrombotic, recent, venous, classical, involved, characterized, infected with SARS-CoV-2, thromboembolic complication, 【제목키워드】 the SARS-CoV-2,