The infectivity of severe acute respiratory syndrome coronavirus 2 in deceased persons and organisms remains unclear. We studied transgenic K18 hACE2 mice to determine the kinetics of virus infectivity after host death. Five days after death, virus infectivity in the lung declined by >96% and RNA copies declined by 48.2%.
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【저자키워드】 COVID-19, 2019 novel coronavirus disease, coronavirus disease, severe acute respiratory syndrome coronavirus 2, viruses, respiratory infections, zoonoses, SARS-CoV-2, Coronaviruses, RT-PCR, Antigen, mice, Postmortem, Infectious virus, Experimental infection, K18 hACE2 mice, TCID50, 【초록키워드】 coronavirus, lung, hACE2, death, Deceased, acute respiratory syndrome, organism, virus infectivity, Host, transgenic, RNA copy, determine, declined, K18, 【제목키워드】 mouse,
【저자키워드】 COVID-19, 2019 novel coronavirus disease, coronavirus disease, severe acute respiratory syndrome coronavirus 2, viruses, respiratory infections, zoonoses, SARS-CoV-2, Coronaviruses, RT-PCR, Antigen, mice, Postmortem, Infectious virus, Experimental infection, K18 hACE2 mice, TCID50, 【초록키워드】 coronavirus, lung, hACE2, death, Deceased, acute respiratory syndrome, organism, virus infectivity, Host, transgenic, RNA copy, determine, declined, K18, 【제목키워드】 mouse,