Potential mechanisms of cerebrovascular diseases in COVID-19 patients
COVID-19 환자의 뇌혈관 질환의 잠재적 기전
Review
[키워드] ACE2
acute respiratory syndrome
acute respiratory syndrome coronavirus
acute respiratory syndrome coronavirus-2
affected
aggravate
Alter
angiotensin
Angiotensin-converting enzyme
angiotensin-converting enzyme 2
antibody
antiphospholipid antibodies
Atherosclerosis
attack
Blood
Blood viscosity
cause
cerebrovascular
Cerebrovascular disease
cerebrovascular diseases
Clinical practice
Coagulation
coronavirus disease
Coronavirus disease 2019
Coronavirus-2
COVID-19
COVID-19 infection
COVID-19 outbreak
COVID-19 patient
COVID-19 patients
CVD
Disruption
dysfunction
Effect
endothelial cells
endothelial damage
enzyme
fibrinolysis
followed by
greater
hemorrhage
hypercoagulability
hyperglycemia
immune
Immune complexes
immunothrombosis
IMPROVE
Inflammation
inflammatory cascade
investigated
involved
leads
mechanism
moment
neurological
neutrophil
Neutrophil extracellular trap
Neutrophil extracellular traps
Occurrence
offered
outbreak
outcome
oxidative stress
patients
plasma
Potential
potential risk
progression
promote
Psychological
Psychological stress
Renin
renin-angiotensin system
respiratory
respiratory manifestations
resulting
risk
SARS-CoV-2
Severe acute respiratory syndrome
severe acute respiratory syndrome Coronavirus
severe cases
Stress
stroke
therapeutic
thrombosis
Treatment
Trigger
upregulation
vascular endothelial cell
vascular endothelial cells
[DOI] 10.1007/s13365-021-00948-2 PMC 바로가기 [Article Type] Review
[DOI] 10.1007/s13365-021-00948-2 PMC 바로가기 [Article Type] Review