Exposure of Candida albicans β (1,3)-glucan is promoted by activation of the Cek1 pathway
Cek1 경로의 활성화에 의해 칸디다 알비칸스 β(1,3)-글루칸의 노출이 촉진됩니다
Research Article
[키워드] activate
activated
Activation
addition
AIDS
antigenic
approach
biogenesis
bloodstream
Cancer patients
Candida
Candida albican
Candida albicans
cascades
cause
Cdc42
Cell
cell wall
cell wall synthesis
decrease
detect
disrupt
Disruption
downstream
drug
exhibit
exposure
Fungal infection
fungal infections
fungal pathogens
glucan
glycosylated
GTPase
homozygous
Hyperactivation
immune
immune cells
immune function
immune system
increase
independent
innate immune cell
Innate immune cells
Intracellular
kinase
kinases
lab
lead
leads
mannan
MAPK
masking
mitogen
Mortality
mutant
Mutation
Pathogens
pathway
Patient
Phosphatidylserine
polysaccharide
promoted
protein kinase
receptor
receptors
regulate
remodeling
responsible
Signaling
signaling cascade
significantly increased
synthesis
the cell
up-regulated
upregulated
upregulation
upstream
virulence
virulence factor
weakened
were used
wild-type
wild-type cell
wild-type cells
[DOI] 10.1371/journal.pgen.1007892 PMC 바로가기 [Article Type] Research Article
[DOI] 10.1371/journal.pgen.1007892 PMC 바로가기 [Article Type] Research Article