Ozone exposure upregulates the expression of host susceptibility protein TMPRSS2 to SARS-CoV-2
Article
[키워드] ACE2
acute infection
Acute inflammation
Affect
airway
airways
alveolar macrophage
Alveolar macrophages
ambient
analyzed
Angiotensin-converting enzyme
binding
cause
cellular receptor
clinical settings
collected
correlated
dependent on
detrimental effect
elevated
Environmental impact
Epidemiology
expression
expression level
health emergency
Host
host cell
host susceptibility
individual
Infection
Interaction
lung tissue
mice
morbidity and mortality
Mortality
pandemic
Pathogenesis
priming
proportion
protease
Protein
proteolytic
respiratory tract
SARS-CoV-2
SARS-CoV-2 entry
Scientific community
serine 2
severe disease
severe SARS-CoV-2
severity
shown
significantly
subsequent
susceptibility
the SARS-CoV-2
TMPRSS2
TMPRSS2 protein
transcript
transmembrane
upregulate
upregulated
virus
while
[DOI] 10.1038/s41598-022-04906-8 PMC 바로가기 [Article Type] Article
[DOI] 10.1038/s41598-022-04906-8 PMC 바로가기 [Article Type] Article