Elevated Cytokine Levels in Plasma of Patients with SARS-CoV-2 Do Not Contribute to Pulmonary Microvascular Endothelial Permeability
SARS-CoV-2 환자의 혈장에서 증가된 사이토카인 수준은 폐 미세혈관 내피 투과성에 기여하지 않습니다
Research Article
[키워드] accounted
ACE-2 receptor
ACE2 antibody
acute respiratory distress
acute respiratory distress syndrome
acute respiratory syndrome
acute respiratory syndrome coronavirus
acute respiratory syndrome coronavirus 2
acute SARS-CoV-2 infection
angiopoietin
antagonists
antiinflammatory cytokines
appear
approach
ARDS
barrier dysfunction
C3a
C5a
cause
Cell
changes in
circulating
Combination
complement
complements factors
consecutive patient
Control
controls
coronavirus
cytokine
Cytokines
death
demonstrated
determine
disrupt
Effect
eight
Elevation
endothelial
Endothelial cell
endothelial injury
endothelial permeability
enrolled
evaluated
Evidence
evidence of
Frequency
greater
growth factor
heat inactivation
human lung
in vitro
infections
inhibitors
labile
Level
lung
magnitude
mechanism
Microvascular damage
morbidity
morbidity and mortality
multiorgan failure
Neutralizing antibodies
neutralizing antibody
occur
patients
patients hospitalized
patients with SARS-CoV-2
permeability
plasma
positive control
predicted
produced
proinflammatory cytokines
provide
pulmonary
receptor
receptor antagonists
respiratory
respiratory distress
SARS-CoV-2
SARS-CoV-2 infected patients
SARS-COV-2 infection
SARS-CoV-2 infections
SARS-CoV-2 patient
SARS-CoV-2 patients
SARS-CoV-2 plasma
selected
severe acute respiratory syndrome Coronavirus
severe complication
severe complications
Spike protein
subsequent
susceptible
syndrome
the SARS-CoV-2
thrombin
TNF-α
Tumor
tumor necrosis
tumor necrosis factor
tumor necrosis factor-alpha
Variability
Vascular
vascular endothelial growth factor
Vascular injury
vascular permeability
VEGF
[DOI] 10.1128/spectrum.01671-21 PMC 바로가기 [Article Type] Research Article
[DOI] 10.1128/spectrum.01671-21 PMC 바로가기 [Article Type] Research Article