LTB 4-Driven Inflammation and Increased Expression of ALOX5/ ACE2 During Severe COVID-19 in Individuals With Diabetes
당뇨병 환자의 중증 COVID-19 기간 동안 LTB 4 유발 염증 및 ALOX5/ACE2 발현 증가
Article
[키워드] ACE2
activate
acute respiratory syndrome
acute respiratory syndrome coronavirus
addition
ALOX5
arterial partial pressure of oxygen
Blood
blood sample
Blood samples
caused
Chronic low-grade inflammation
Clinical data
collected
coronavirus
coronavirus disease
Coronavirus disease 2019
COVID-19
diabete
diabetes
disease
disorders
evaluate
Evolution
expression
expressions
Gene expression analysis
Hospitalized
IL-6
increase
Increased
increased expression
individual
inflammatory mediator
Inflammatory mediators
intensive care
involved
knowledge
lack
LTB
main receptor
marker
mechanism
molecular
mononuclear cells
New coronavirus
oxygen
oxygen saturation
partial pressure
pathway
Patient
patients with diabete
Peripheral blood
Peripheral blood mononuclear cells
plasma
plasma samples
positively correlated
protein level
pulse
pulse oximetry
reduced
risk factor
SARS-CoV-2
Serum level
serum levels
severe coronavirus disease
severe COVID-19
significantly increased
the disease
the SARS-CoV-2
TMPRSS2
were used
[DOI] 10.2337/db20-1260 PMC 바로가기 [Article Type] Article
[DOI] 10.2337/db20-1260 PMC 바로가기 [Article Type] Article