COVID-19 lung disease shares driver AT2 cytopathic features with Idiopathic pulmonary fibrosis
COVID-19 폐 질환은 특발성 폐 섬유증과 드라이버 AT2 세포 변성 기능을 공유합니다
Article
[키워드] alveolar
Alveolar Type II
Alveolar type II pneumocytes
Analysis
analyzed
approach
artificial
Artificial intelligence/machine learning
AT2
cells
change
changes
condition
COVID
COVID-19
Cytokine storm
cytopathic
Damage-associated transient progenitor (DATP)
dataset
Deceased
determinant
develop
disease
Disease model
disease models
Diseases
DNA damage
driven by
driver
dysfunction
effective
ER stress
ER-stress
feature
gene expression pattern
gene signatures
hamsters
Host immune response
human lung
identify
Idiopathic pulmonary fibrosis
IHC
immune response
immunohistochemistry
Injury
interleukin
Interleukin 15 (IL15)
Interstitial lung diseases
IPF
lack
Like
lung
Lung disease
Lungs
molecular
monocyte
pandemic
Pathogenesis
Patient
patients with IPF
Perspective
phenotype
PPI
Prognosis
prognostic
Program
protein-protein interaction
pulmonary fibrosis
qPCR
reversed
SARS-CoV-2
SASP
senescence
similarity
subject
supported
therapy
transcriptomic
Treatment
Trigger
were used
with COVID-19
[DOI] 10.1016/j.ebiom.2022.104185 PMC 바로가기 [Article Type] Article
[DOI] 10.1016/j.ebiom.2022.104185 PMC 바로가기 [Article Type] Article