multiorgan failure
[용어속성] Symptom
The potential role of COVID-19 in the induction of DNA damage
DNA 손상 유도에서 COVID-19의 잠재적 역할
Review
Published on
Journal: Mutation research. Reviews in mutation research [Category] SARS, 변종, 유전자 메커니즘,
Journal: Mutation research. Reviews in mutation research [Category] SARS, 변종, 유전자 메커니즘,
[키워드] acute respiratory syndrome
acute respiratory syndrome coronavirus
acute respiratory syndrome coronavirus-2
affecting
appearance
Atherosclerosis
Brain
Cancer
caused
Cell
cell cycle
chronic disease
Chronic diseases
coronavirus
coronavirus disease
Coronavirus-2
Coronaviruses
COVID-19
Cytokine storm syndrome
death
deregulation
diabetes
direct viral infection
DNA
DNA damage
DNA repair
DNA repair mechanism
Effect
Evidence
evidence of
genomic
Health
heart
immunological
in some
individuals
induce
kidney
kidneys
Lungs
mammalian cells
mechanism
multiorgan failure
neurodegenerative disorders
organs
outcome
Recovered COVID-19 patients
recovered patient
recovered patients
Replication
SARS-CoV-2
Sepsis
Severe acute respiratory syndrome
severe acute respiratory syndrome Coronavirus
severe acute respiratory syndrome coronavirus-2
Support
Symptoms
these viruses
viral infection
virus
[DOI] 10.1016/j.mrrev.2022.108411 PMC 바로가기 [Article Type] Review
[DOI] 10.1016/j.mrrev.2022.108411 PMC 바로가기 [Article Type] Review
A virus-specific monocyte inflammatory phenotype is induced by SARS-CoV-2 at the immune–epithelial interface
420
[키워드] acute respiratory syndrome
age
B cells
Blood
children
Coronavirus-2
COVID-19
COVID-19 patients
cross-talk
Cytokine storm
deviation
Ebola
elicit
encodes
epithelia
epithelial
epithelial cell
Express
Factors
fraction
gene cluster
hallmark
healthy children
Immune cell
immune cells
immune dysregulation
induce
infected with SARS-CoV-2
Inflammatory
inflammatory phenotype
influenza A
initial
Interaction
interferon
lung
manifestation
monocyte
multiorgan failure
myeloid cell
nonstructural proteins
pathogenic viruses
Patient
Perturbation
Pneumonia
Protein
provide
provoke
reported
SARS-CoV-2
SARS-CoV-2 protein
severe COVID-19
severe disease
Significance
spike
systemic inflammation
transcriptional
transduced
virus
[DOI] 10.1073/pnas.2116853118 PMC 바로가기 [Article Type] 420
[DOI] 10.1073/pnas.2116853118 PMC 바로가기 [Article Type] 420
Clinical Management of COVID-19 in Cancer Patients with the STAT3 Inhibitor Silibinin
Perspective
[키워드] active cancer
acute respiratory distress
addition
Admission
ARDS
arising
C-reactive protein
cascade
caused
clinical
clinical trial
Complete
COVID-19
COVID-19 illness
COVID-GRAM
COVID-GRAM score
Critical
death
demonstrated
disease
dysregulation
EudraCT
events
exhibited
high risk
hospitalized patient
IMPROVE
inhibitor
intensive care
interstitial pneumonia
Intervention
invasive ventilation
knowledge
Legalon
lymphocytopenia
multiorgan failure
outcome
oxygen support
Pathogenesis
pathophysiology
Patient
patients
Perspective
predicted
presenting
Proinflammatory response
pulmonary lesions
receiving
recruited
required
Research
SARS-CoV-2
severe COVID-19
Side-effects
silibinin
STAT3
supportive care
syndrome
therapy
transcriptional
treating COVID-19
Treatment
[DOI] 10.3390/ph15010019 PMC 바로가기 [Article Type] Perspective
[DOI] 10.3390/ph15010019 PMC 바로가기 [Article Type] Perspective
COVID-19 and Sepsis
Article
[키워드] acute phase reactant
acute respiratory distress
addition
affecting
alterations
ARDS
assist
association
Autopsy
Bacterial sepsis
Blood
cause
caused
Cell
changes
chemokine
Clinical deterioration
coagulation tests
component
consolidation
contributing to
Course
COVID-19
COVID-19 pandemic
COVID-19 pathogenesis
COVID-19 patient
COVID-19 sepsis
CRP
Cytokine storm
D-dimer
demonstrated
Diffuse alveolar damage
direct viral infection
dominant
dysregulated immune response
endothelial injury
explained
Faeces
feature
ferritin
Gallbladder
hematological
hemorrhage
histopathological
HLH
hospitalized patient
Hyperactivation
hypercoagulability
hypercoagulable
hyperinflammation
immune
immune response
IMPROVE
indicated
infiltration
inflammatory cell
intrinsic
Laboratory
laboratory parameter
limit
liver
liver function tests
lung tissue
Lungs
macrophages
manifestation
modulate
multiorgan failure
multiple organ
organ systems
organs
outcome
overlapping
pandemic
Pathogenesis
pathogenesis of COVID-19
Pathogens
Result
SARS-CoV-2
Sepsis
serum cytokine
Severe case
severe COVID-19
severe COVID-19 disease
Severe COVID-19 Infection
severe COVID-19 patients
similarity
syndrome
systemic inflammation
tissue
Treatment
unique
Urine
Vascular
viral disease
viral particle
virus
while
[DOI] 10.3906/sag-2108-239 PMC 바로가기 [Article Type] Article
[DOI] 10.3906/sag-2108-239 PMC 바로가기 [Article Type] Article
Massive image-based single-cell profiling reveals high levels of circulating platelet aggregates in patients with COVID-19
Article
[키워드] aggregate
aggregation
Analysis
arteriole
Autopsy
Blood
characteristic
characterized
circulating platelet
Concentration
COVID-19
COVID-19 patient
COVID-19 patients
Diagnostic markers
Endothelial dysfunction
flow cytometry
hearts
imaging
incidence
Lab-on-a-chip
lack
Lungs
massive
microthrombi
microthrombus
Microvascular thrombosis
Mortality
multiorgan failure
other organs
patients with COVID-19
peripheral capillary
Platelet
resulting
reveal
severity
shown
single-cell
statistically
thrombotic
Vascular
widespread
[DOI] 10.1038/s41467-021-27378-2 PMC 바로가기 [Article Type] Article
[DOI] 10.1038/s41467-021-27378-2 PMC 바로가기 [Article Type] Article
Extracorporeal carbon dioxide removal with the Advanced Organ Support system in critically ill COVID-19 patients
Observational Study
[키워드] acid-base balance
Acute kidney injury
acute respiratory distress
ADVOS
AKI
analyzed
approach
ARDS
baseline
Berlin
blood flow
CO2
Cohort
correlated
COVID-19
COVID-19 patient
COVID-19 patients
Critically ill
develop
Disturbed
eligible
evaluate
Extracorporeal
extracorporeal CO2 removal (ECCO2R)
feasible
Germany
HCO
Hemodialysis
invasive
Invasive mechanical ventilation
KDIGO
kidney
less
liver
lung
median
membrane
moderate
multi
multi-organ failure
multiorgan failure
multiple organ support.
Munich
nine
organ support
Oxygenation
Patient
Pearson
problem
problems
proportion
receiving
reduction in
severe ARDS
significantly
study inclusion
Support System
syndrome
therapy
Treatment
[DOI] 10.1111/aor.14044 PMC 바로가기 [Article Type] Observational Study
[DOI] 10.1111/aor.14044 PMC 바로가기 [Article Type] Observational Study
Anakinra as a potential alternative in the treatment of severe acute respiratory infection associated with SARS-CoV-2 refractory to tocilizumab
토실리주맙에 불응성인 SARS-CoV-2와 관련된 중증 급성 호흡기 감염 치료의 잠재적인 대안으로서의 아나킨라
Case Reports
[키워드] (Tocilizumab
Acute respiratory infection
Anakinra
anti-IL-1
anti-IL-6
anti-IL-6 receptor
anti-IL-6 treatment
antibodies
Antiviral
Atypical
Atypical pneumonia
Case report
cause
Clinical outcome
coronavirus disease
Coronavirus disease 2019
COVID-19
cytokine
Cytokine storm
diagnosed
dysregulation
English
haemophagocytic lymphohistiocytosis
Health Organization
IL-1
IL-6
immune system
Inflammatory response
multiorgan failure
OMS
Pandemia
pandemic
Pneumonia
Produce
receptor
respiratory
respiratory infection
RNA virus
SARS-CoV-2
severe respiratory infection
Spanish
Tocilizumab
Tormenta de citoquinas.
Treatment
Trigger
triggering
triggers
virus
virus RNA
WHO
World Health Organization
[DOI] 10.1016/j.reumae.2020.06.008 PMC 바로가기 [Article Type] Case Reports
[DOI] 10.1016/j.reumae.2020.06.008 PMC 바로가기 [Article Type] Case Reports
A review of acute pancreatitis in the era of COVID-19
Review article
[키워드] acute pancreatitis
approach
association
childhood
children
collected
Complication
Course
COVID-19
Diagnosis
disease
disease pathogenesis
Epidemiology
Evidence
genetics
high risk
Hospital admission
incidence
Intervention
invasive
management
mild illness
morbidity and mortality
Mortality
MOST
multiorgan failure
occurred
paediatric pancreatitis
pathophysiology
Prognosis
progression
resulting
severe disease
significantly
suggested
[DOI] 10.1016/j.paed.2021.09.001 [Article Type] Review article
[DOI] 10.1016/j.paed.2021.09.001 [Article Type] Review article
Identification of Cellular Factors Required for SARS-CoV-2 Replication
Article
[키워드] ACE2
acute respiratory syndrome
Analysis
angiotensin-converting enzyme 2
Antiviral agents
Asymptomatic disease
cellular factors
clinical
complex
coronavirus
COVID-19 pandemic
CRISPR-Cas9
cross
death
entry receptor
Factor
HeLa cell
host cell
host–virus interaction
Human
identification
include
infect
Infection
insight
Interaction
knock-out
Lung injury
mechanisms of infection
mild respiratory
multifactorial
multiorgan failure
Organisms
overexpressing
platform
Prevent
receptors
replicate
replication mechanism
required
responsible
resulting
SARS-CoV-2
SARS-COV-2 infection
severe disease
unique
viral pathogenesis
viral replication
virus
[DOI] 10.3390/cells10113159 PMC 바로가기 [Article Type] Article
[DOI] 10.3390/cells10113159 PMC 바로가기 [Article Type] Article