Novel Coronavirus Disease 2019 (COVID-19) and Cytokine Storms for More Effective Treatments from an Inflammatory Pathophysiology
Review
[키워드] Abnormalities
ACE2
activate
acute respiratory distress
Administered
Against
anti-rheumatic drug
bat
cause
caused
Cell
cell debris
Central nervous system
chemokine
chemokines
China
chronic inflammation
Coagulation
COVID-19
cytokine
cytokine receptor
Cytokine release syndrome
Cytokine storm
Damage-associated molecular patterns
develop
disorders
Disseminated intravascular coagulation
DNA
Effect
endothelial cell damage
Europe
excessive inflammatory response
Favipiravir
G-CSF
global pandemic
Host
human Angiotensin-converting enzyme
IL-1β
IL-6
IL-8
immune system
Infection
Infectious disease
Inflammatory response
inhibit
innate immune system
Innate immunity
involved
IP10
liver
melatonin
mitochondrial
mitochondrial dysfunction
Mitochondrial function
monoclonal antibodies
multiple organ failure
new virus
novel
novel coronavirus diseases (or COVID-19)
occur
organ damage
patients
producing
Proinflammatory cytokine
renal
respiratory infection
resulting
SARS-CoV
SARS-CoV-2
second
serum
storm
Stress
subject
subsequent
suppress
syndrome
TNF-α
Tocilizumab
treatment for COVID-19
Vascular
viral invasion
[DOI] 10.3390/jcm10040801 PMC 바로가기 [Article Type] Review
[DOI] 10.3390/jcm10040801 PMC 바로가기 [Article Type] Review