A type I IFN, prothrombotic hyperinflammatory neutrophil signature is distinct for COVID-19 ARDS
Research Article
[키워드] accumulating
activated
Activation
acute respiratory syndrome
aggregate
antigen processing
ARDS
binding
cascade
caused
characteristic
circulating
clinical entity
complex
component
coronavirus
coronavirus disease
COVID-19
COVID-19 patients
critical condition
Dexamethasone
distress
dysregulation
Evidence
expansion
functional
high mortality
Hyperinflammatory
immature
immature neutrophil
Infection
infection with SARS-CoV-2
Lung injury
metabolism
metabolomic
molecular basis
Mortality
neutrophil
neutrophil activation
Non-COVID-19
occur
Patient
patients with COVID-19
Platelet
Population
populations
Proteins
proteomic
prothrombotic
Release
response
SARS-CoV-2
syndrome
Treatment
Type I IFN
type I interferon
upregulation
[DOI] 10.12688/wellcomeopenres.16584.2 PMC 바로가기 [Article Type] Research Article
[DOI] 10.12688/wellcomeopenres.16584.2 PMC 바로가기 [Article Type] Research Article