A Bittersweet Response to Infection in Diabetes; Targeting Neutrophils to Modify Inflammation and Improve Host Immunity
Immunology
[키워드] abnormality
biochemical
breadth
chemotaxis
chronic
complex
COVID-19
Critical
diabete
diabetes
disease
dysregulated
Extracellular
Future
handling
highlighting
Host
host immunity
hyperglycaemia
immune response
IMPROVE
increased risk
individual
Infection
Inflammation
innate immune system
mechanism
morbidity and mortality
NET
NETosis
neutrophil
neutrophil dysfunction
Neutrophil extracellular trap
neutrophil function
neutrophil recruitment
occur
pathogen
Pathways
Patient
phagocyte
phagocytosis
phenotype
pro-inflammatory
pro-inflammatory cytokine
proinflammatory
reactive oxygen specy
recurrent infection
Research
response
ROS
severe coronavirus disease
significantly
T1D
T2D
targeting
therapy
Treatment
Type 1 diabetes
type 2 diabete
type 2 diabetes
upregulated
[DOI] 10.3389/fimmu.2021.678771 PMC 바로가기 [Article Type] Immunology
[DOI] 10.3389/fimmu.2021.678771 PMC 바로가기 [Article Type] Immunology