pro-inflammatory cytokine – Page 23 – Communicable Diseases Research

Anti-Rheumatic Drugs for the Fight Against the Novel Coronavirus Infection (SARSCoV-2): What is the Evidence?

Review

Published on 2020-09-212022-10-31
Journal: Mediterranean Journal of Rheumatology [Category] COVID-19,

The Potential Role of SARS-COV-2 in the Pathogenesis of Parkinson’s Disease

Neurology

Published on 2020-09-172022-11-01
Journal: Frontiers in Neurology [Category] COVID-19,

[키워드] accompany affected alpha-synuclein Autoimmune reaction Autopsy Brain Cell change coronavirus COVID-19 pandemic cross CSF demonstrated disease epidemiological Evidence ganglia Health Immunologic Inflammatory response Movement disorders nervous system Nervous system diseases neurodegeneration neurodegenerative Neurodegenerative diseases neurotropic Pandemics Parkinson disease Pathogenesis pathogenic Patient patients patients with COVID-19 Potential pro-inflammatory cytokine pro-inflammatory state problems Prognostic factor progression role SARS virus SARS-CoV-2 SARS-COV-2 infection triggers viral infectious disease virus
[DOI] 10.3389/fneur.2020.01044 PMC 바로가기 [Article Type] Neurology

The efficacy of colchicine in the management of coronavirus disease 2019 A protocol for systematic review and meta-analysis

4900

Published on 2020-09-042022-10-30
Journal: Medicine [Category] COVID-19, SARS,

[키워드] absence acute respiratory distress acute respiratory syndrome anti-inflammatory agent anticipated ARDS chemokines Colchicine coronavirus coronavirus disease Coronavirus disease 2019 COVID-19 COVID-19 patients COVID-19 related mortality disease dysregulated dysregulated response effective Efficacy Efficacy and safety imperative include Infection Inflammatory response instances management Meta-analysis Mild mitigate Mortality mortality risk outcome outcomes peer-reviewed primary endpoint pro-inflammatory cytokine protocol random-effect Randomized controlled trial reducing severe acute respiratory syndrome coronavirus 2 syndrome systematic review therapeutic option therapeutic options upper respiratory symptoms viral infection
[DOI] 10.1097/MD.0000000000021911 PMC 바로가기 [Article Type] 4900

Cytokine release syndrome: inhibition of pro-inflammatory cytokines as a solution for reducing COVID-19 mortality

사이토카인 방출 증후군: COVID-19 사망률을 줄이기 위한 솔루션으로 전염증성 사이토카인 억제

Review

Published on 2020-09-012022-09-12
Journal: European cytokine network [Category] SARS, 진단,

COVID-19 and ethnicity: A novel pathophysiological role for inflammation

Research article

Published on 2020-09-012022-10-05
Journal: Diabetes & metabolic syndrome [Category] SARS, 유전자 메커니즘,

[키워드] Acute inflammation appear article articles Bame black can be used cardiovascular disease cardiovascular risk chronic disease chronic infection chronic inflammation chronic systemic inflammation contributing to COVID-19 COVID-19 mortality COVID-19 research Cytokine storm database disease severity dysregulation elevated English Ethnicity explain Factor Genetic predisposition Google Scholar identify Inflammation insulin resistance management metabolic syndrome National obesity pathophysiological pathophysiological mechanism performed phenotype pro-inflammatory cytokine provide Psychological Psychological stress Research Result risk SARS CoV-2 searched severe COVID-19 disease severe disease significantly Stress Type 2 diabetes mellitus visceral fat
[DOI] 10.1016/j.dsx.2020.06.056 [Article Type] Research article

Influence of NAD+ as an ageing-related immunomodulator on COVID 19 infection: A hypothesis

Review article

Published on 2020-09-012022-10-05
Journal: Journal of infection and public health [Category] SARS, 유전자 메커니즘,

[키워드] addition adenine Affect Ageing alteration antigen-presenting cell biological function Cellular immune response change chromosome circulating pro-inflammatory cytokine Complication COVID 19 COVID-19 COVID-19 patient Cytokine storm death dinucleotide Effects elderly Factor Gas exchange Hospitalization Hypothesis immune Immune cell immune function immune system immunomodulator impairment increase in Increasing influence inhibitory effect lung lymphocyte morbidity and mortality NAD+ positive Prevent pro-inflammatory cytokine Respiratory function risk sensitivity Severe infection stability susceptibility telomere telomeres
[DOI] 10.1016/j.jiph.2020.06.004 [Article Type] Review article

Baricitinib, a drug with potential effect to prevent SARS-COV-2 from entering target cells and control cytokine storm induced by COVID-19

Review article

Published on 2020-09-012022-10-05
Journal: International immunopharmacology [Category] SARS, 치료제,

[키워드] ACE2 ACE2 receptor acute respiratory syndrome Angiotensin-converting enzyme atopic dermatitis Baricitinib C reactive protein Care caused causes Cell cells China clinical benefit Clinical characteristics clinical trial coronavirus COVID-19 CRP cytokine Cytokine storm demonstrated Deterioration develop disease Disease progression drug Efficacy and safety expected inhibit intracellular assembly involved Jak Jak-STAT JAK-STAT pathway Janus kinase Lymphocytes mechanism much lower natural killer Novel coronavirus Patient Pharmaceutical care Pneumonia Prevent pro-inflammatory cytokine proinflammatory reducing reported Research rheumatoid arthritis SARS-CoV-2 SARS-CoV-2 entry severe COVID-19 severe pneumonia Spread surface protein systemic lupus erythematosus target cell the cytokine storm the patient treat Treatment US FDA viral entry
[DOI] 10.1016/j.intimp.2020.106749 [Article Type] Review article

Aryl Hydrocarbon Receptor Role in Co-Ordinating SARS-CoV-2 Entry and Symptomatology: Linking Cytotoxicity Changes in COVID-19 and Cancers; Modulation by Racial Discrimination Stress

Review

Published on 2020-08-272022-10-28
Journal: Biology [Category] COVID-19,

[키워드] acetyl-CoA activate Anti-viral anti-viral response aryl Aryl hydrocarbon receptor Asian Bame black Cancer cancers cells change contribute COVID-19 COVID-19 pandemic COX cytolytic discrimination fatality Gut Health hydrocarbon IDO immune Immune cell implication increase in Indoleamine 2,3-dioxygenase induce initial Medical conditions melatonin Mitochondria mitochondrial metabolism modulating pathophysiological pathophysiology pathway Pathways pro-inflammatory cytokine Prophylactic Racial racism raised regulated Research role SARS-CoV-2 SARS-COV-2 infection severity Stress Treatment Type II Diabetes
[DOI] 10.3390/biology9090249 PMC 바로가기 [Article Type] Review

Analysis of Co-inhibitory Receptor Expression in COVID-19 Infection Compared to Acute Plasmodium falciparum Malaria: LAG-3 and TIM-3 Correlate With T Cell Activation and Course of Disease

급성 열대열 말라리아와 비교한 COVID-19 감염의 공동 억제 수용체 발현 분석: LAG-3 및 TIM-3은 T 세포 활성화 및 질병 경과와 상관관계가 있습니다

Immunology

Published on 2020-08-262022-11-01
Journal: Frontiers in Immunology [Category] COVID-19, MERS, SARS, 말라리아,

[키워드] Activation acute COVID-19 B cell response BTLA cause caused CCR7 CD226 CD38 CD4 CD45RO CD69 CD8 characterized co-expression Coronavirus disease 2019 COVID-19 COVID-19 patient disease course effector cell elevated Express expression expression pattern Frequency healthy controls HLA-DR hyperinflammation Hypothesis immune immune activation immune dysfunction Immunoglobulin implicated induce infected patient Infection inhibitory receptor LAG-3 limit malaria Mild novel SARS-CoV-2 virus outcome P. falciparum pathology Patient PBMC PD-1 PD1 Plasmodium falciparum Plasmodium falciparum malaria populations pro-inflammatory cytokine rapid normalization receptor SARS-CoV-2 SARS-CoV-2 patient severe disease course T cell T cells T-cell thought TIGIT TIM-3 transcription factor upregulation variety viral infection virus
[DOI] 10.3389/fimmu.2020.01870 PMC 바로가기 [Article Type] Immunology

Implication of Aging Related Chronic Neuroinflammation on COVID-19 Pandemic

Review

Published on 2020-08-262022-10-30
Journal: Journal of Personalized Medicine [Category] COVID-19,

[키워드] affected aging Brain brain inflammation characterized chronic chronic inflammation clinician complications of COVID-19 condition contribute COVID-19 COVID-19 neurological and psychiatric manifestations COVID-19 pandemic COVID-19 patients death Diseases disorders dysregulation elderly elevated frailty general condition help Hyperactivation implication increase in increases in innate immune innate immune system involved low-grade inflammation Manifestations mechanism mild symptoms mortality risk MOST neurodegenerative Neuroinflammation Neuropsychiatric neuropsychiatric condition occur Older outcome pandemic pathway Patient pro-inflammatory cytokine responsible risk SARS-CoV-2 selected Severe case severe COVID-19 severe disease syndrome the disease triggered virus virus infection worsened
[DOI] 10.3390/jpm10030102 PMC 바로가기 [Article Type] Review